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Acta Neuropathologica
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1. Dissecting the genetic basis of focal cortical dysplasia: a large cohort study
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Tác giả
Sara Baldassari, Théo Ribierre, Elise Marsan, Homa Adle-Biassette.
Nội dung
Pages 885-900
1. On the journey to uncover the causes of selective cellular and regional vulnerability in neurodegeneration
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Tác giả
Zane Jaunmuktane, Sebastian Brandner.
Nội dung
Pages 677-680
1. Calling α-synuclein a prion is scientifically justifiable
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Tác giả
Joel C. Watts.
Nội dung
Pages 505-508
1. Neurotoxicology: an update on epidemiology, mechanisms, and pathology
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Tác giả
Christoph van Thriel.
Nội dung
Pages 339-341
1. Novel Alzheimer’s disease risk genes: exhaustive investigation is paramount
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Tác giả
Kristel Sleegers, Christine Van Broeckhoven.
Nội dung
Pages 171-172
1. α-Synuclein and astrocytes: tracing the pathways from homeostasis to neurodegeneration in Lewy body disease
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Tác giả
Zachary A. Sorrentino, Benoit I. Giasson, Paramita Chakrabarty.
Nội dung
Pages 1-21
1. Disrupted neuronal trafficking in amyotrophic lateral sclerosis
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Tác giả
Katja Burk, R. Jeroen Pasterkamp.
Nội dung
Pages 859-877
1. Neuroinflammation, the thread connecting neurological disease
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Tác giả
Roberta Brambilla.
Nội dung
Pages 689-691
1. Mission not yet completed: on the ups and downs of being the editor of ANP
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Tác giả
Werner Paulus.
Nội dung
Pages 531-534
1. Renewed assessment of the risk of emergent advanced cell therapies to transmit neuroproteinopathies
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Tác giả
Paul A. De Sousa, Diane Ritchie, Alison Green.
Nội dung
Pages 363-377
1. The role of de novo mutations in adult-onset neurodegenerative disorders
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Tác giả
Gaël Nicolas, Joris A. Veltman.
Nội dung
Pages 183-207
1. C9orf72-FTD/ALS pathogenesis: evidence from human neuropathological studies
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Tác giả
Sarat C. Vatsavayai, Alissa L. Nana, Jennifer S. Yokoyama.
Nội dung
Pages 1-26
2. Precise detection of low-level somatic mutation in resected epilepsy brain tissue
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Tác giả
Nam Suk Sim, Ara Ko, Woo Kyeong Kim, Se Hoon Kim, Ju Seong Kim.
Nội dung
Pages 901-912
2. Selective vulnerability in α-synucleinopathies
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Tác giả
Javier Alegre-Abarrategui, Katherine R. Brimblecombe.
Nội dung
Pages 681-704
2. Alpha-synuclein: prion or prion-like?
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Tác giả
Rehana K. Leak, Matthew P. Frosch, Thomas G. Beach.
Nội dung
Pages 509-514
2. Neurotoxicity of pesticides
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Tác giả
Jason R. Richardson, Vanessa Fitsanakis, Remco H. S. Westerink.
Nội dung
Pages 343-362
2. SORL1 genetic variants and Alzheimer disease risk: a literature review and meta-analysis of sequencing data
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Tác giả
Dominique Campion, Camille Charbonnier, Gaël Nicolas.
Nội dung
Pages 173-186
2. Binding of α-synuclein oligomers to Cx32 facilitates protein uptake and transfer in neurons and oligodendrocytes
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Tác giả
Juan F. Reyes, Christopher Sackmann, Alana Hoffmann.
Nội dung
Pages 23-47
2. Genome-wide analyses as part of the international FTLD-TDP whole-genome sequencing consortium reveals novel disease risk factors and increases support for immune dysfunction in FTLD
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Tác giả
Cyril Pottier, Yingxue Ren, Ralph B. Perkerson III, Matt Baker.
Nội dung
Pages 879-899
2. Post-stroke inflammation—target or tool for therapy?
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Tác giả
Kate Lykke Lambertsen, Bente Finsen, Bettina Hjelm Clausen.
Nội dung
Pages 693-714
2. Translational control in brain pathologies: biological significance and therapeutic opportunities
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Tác giả
Alberto Delaidelli, Asad Jan, Jochen Herms, Poul H. Sorensen.
Nội dung
Pages 535-555
2. Retinal α-synuclein deposits in Parkinson’s disease patients and animal models
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Tác giả
Lien Veys, Marjan Vandenabeele, Isabel Ortuño-Lizarán.
Nội dung
Pages 379-395
2. Dissecting the genetic relationship between cardiovascular risk factors and Alzheimer’s disease
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Tác giả
Iris J. Broce, Chin Hong Tan, Chun Chieh Fan, Iris Jansen.
Nội dung
Pages 209-226
2. Neurons selectively targeted in frontotemporal dementia reveal early stage TDP-43 pathobiology
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Tác giả
Alissa L. Nana, Manu Sidhu, Stephanie E. Gaus, Ji-Hye L. Hwang.
Nội dung
Pages 27-46
3. Aβ-induced acceleration of Alzheimer-related τ-pathology spreading and its association with prion protein
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Tác giả
Luis Aragão Gomes, Silvia Andrea Hipp, Ajeet Rijal Upadhaya.
Nội dung
Pages 913-941
3. Cellular and regional vulnerability in frontotemporal tauopathies
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Tác giả
Shelley L. Forrest, Jillian J. Kril, Glenda M. Halliday.
Nội dung
Pages 705-727
3. Oral and intravenous transmission of α-synuclein fibrils to mice
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Tác giả
Stephanie Lohmann, Maria E. Bernis, Babila J. Tachu.
Nội dung
Pages 515-533
3. Neurotoxicity of polychlorinated biphenyls and related organohalogens
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Tác giả
Isaac N. Pessah, Pamela J. Lein, Richard F. Seegal.
Nội dung
Pages 363-387
3. Evaluation of CD33 as a genetic risk factor for Alzheimer’s disease
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Tác giả
Steven Estus, Benjamin C. Shaw, Nicholas Devanney.
Nội dung
Pages 187-199
3. ADAR2 mislocalization and widespread RNA editing aberrations in C9orf72-mediated ALS/FTD
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Tác giả
Stephen Moore, Eric Alsop, Ileana Lorenzini, Alexander Starr.
Nội dung
Pages 49-65
3. Loss of DPP6 in neurodegenerative dementia: a genetic player in the dysfunction of neuronal excitability
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Tác giả
Rita Cacace, Bavo Heeman, Sara Van Mossevelde, Arne De Roeck.
Nội dung
Pages 901-918
3. Inflammation in ALS/FTD pathogenesis
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Tác giả
Madelyn E. McCauley, Robert H. Baloh.
Nội dung
Pages 715-730
3. Integrated DNA methylation and gene expression profiling across multiple brain regions implicate novel genes in Alzheimer’s disease
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Tác giả
Stephen A. Semick, Rahul A. Bharadwaj, Leonardo Collado-Torres.
Nội dung
Pages 557-569
3. Amyloid-β pathology enhances pathological fibrillary tau seeding induced by Alzheimer PHF in vivo
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Tác giả
Cristina Vergara, Sarah Houben, Valérie Suain, Zehra Yilmaz.
Nội dung
Pages 397-412
3. Pathological, imaging and genetic characteristics support the existence of distinct TDP-43 types in non-FTLD brains
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Tác giả
Keith A. Josephs, Melissa E. Murray, Nirubol Tosakulwong.
Nội dung
Pages 227-238
3. Mitochondria, ER, and nuclear membrane defects reveal early mechanisms for upper motor neuron vulnerability with respect to TDP-43 pathology
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Tác giả
Mukesh Gautam, Javier H. Jara, Nuran Kocak, Lauren E. Rylaarsdam.
Nội dung
Pages 47-69
4. Granulovacuolar degeneration bodies are neuron-selective lysosomal structures induced by intracellular tau pathology
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Tác giả
Vera I. Wiersma, Anna Maria van Ziel, Sonia Vazquez-Sanchez.
Nội dung
Pages 943-970
4. The basis of cellular and regional vulnerability in Alzheimer’s disease
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Tác giả
Dunja Mrdjen, Edward J. Fox, Syed A. Bukhari, Kathleen S. Montine.
Nội dung
Pages 729-749
4. Evidence for bidirectional and trans-synaptic parasympathetic and sympathetic propagation of alpha-synuclein in rats
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Tác giả
Nathalie Van Den Berge, Nelson Ferreira, Hjalte Gram.
Nội dung
Pages 535-550
4. Chronic traumatic encephalopathy is a common co-morbidity, but less frequent primary dementia in former soccer and rugby players
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Tác giả
Edward B. Lee, Kevin Kinch, Victoria E. Johnson.
Nội dung
Pages 389-399
4. The role of ABCA7 in Alzheimer’s disease: evidence from genomics, transcriptomics and methylomics
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Tác giả
Arne De Roeck, Christine Van Broeckhoven, Kristel Sleegers.
Nội dung
Pages 201-220
4. FUS pathology in ALS is linked to alterations in multiple ALS-associated proteins and rescued by drugs stimulating autophagy
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Tác giả
Lara Marrone, Hannes C. A. Drexler, Jie Wang, Priyanka Tripathi.
Nội dung
Pages 67-84
4. Caspase-4 mediates cytoplasmic accumulation of TDP-43 in the primate brains
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Tác giả
Peng Yin, Xiangyu Guo, Weili Yang, Sen Yan, Su Yang, Ting Zhao.
Nội dung
Pages 919-937
4. The complexity of neuroinflammation consequent to traumatic brain injury: from research evidence to potential treatments
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Tác giả
Maria Cristina Morganti-Kossmann, Bridgette D. Semple.
Nội dung
Pages 731-755
4. Tau drives translational selectivity by interacting with ribosomal proteins
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Tác giả
Shon A. Koren, Matthew J. Hamm, Shelby E. Meier, Blaine E. Weiss.
Nội dung
Pages 571-583
4. Frontal cortex and striatal cellular and molecular pathobiology in individuals with Down syndrome with and without dementia
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Tác giả
Sylvia E. Perez, Jennifer C. Miguel, Bin He, Michael Malek-Ahmadi.
Nội dung
Pages 413-436
4. The metalloprotease ADAMTS4 generates N-truncated Aβ4–x species and marks oligodendrocytes as a source of amyloidogenic peptides in Alzheimer’s disease
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Tác giả
Susanne Walter, Thorsten Jumpertz, Melanie Hüttenrauch.
Nội dung
Pages 239-257
4. Rare variants in the neuronal ceroid lipofuscinosis gene MFSD8 are candidate risk factors for frontotemporal dementia
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Tác giả
Ethan G. Geier, Mathieu Bourdenx, Nadia J. Storm.
Nội dung
Pages 71-88
5. Early defects in translation elongation factor 1α levels at excitatory synapses in α-synucleinopathy
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Tác giả
Sonja Blumenstock, Maria Florencia Angelo, Finn Peters.
Nội dung
Pages 971-986
5. The basis of clinicopathological heterogeneity in TDP-43 proteinopathy
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Tác giả
Ito Kawakami, Tetsuaki Arai, Masato Hasegawa.
Nội dung
Pages 751-770
5. Tau is required for progressive synaptic and memory deficits in a transgenic mouse model of α-synucleinopathy
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Tác giả
Balvindar Singh, Ana Covelo, Héctor Martell-Martínez.
Nội dung
Pages 551-574
5. Contact sport participation and chronic traumatic encephalopathy are associated with altered severity and distribution of cerebral amyloid angiopathy
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Tác giả
Oliver J. Standring, Jacob Friedberg, Yorghos Tripodis.
Nội dung
Pages 401-413
5. The new genetic landscape of Alzheimer’s disease: from amyloid cascade to genetically driven synaptic failure hypothesis?
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Tác giả
Pierre Dourlen, Devrim Kilinc, Nicolas Malmanche, Julien Chapuis.
Nội dung
Pages 221-236
5. The molecular pathogenesis of superoxide dismutase 1-linked ALS is promoted by low oxygen tension
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Tác giả
Isil Keskin, Elin Forsgren, Manuela Lehmann, Peter M. Andersen.
Nội dung
Pages 85-101
5. Alpha-synuclein suppresses mitochondrial protease ClpP to trigger mitochondrial oxidative damage and neurotoxicity
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Tác giả
Di Hu, Xiaoyan Sun, Xudong Liao, Xinwen Zhang, Sara Zarabi.
Nội dung
Pages 939-960
5. The contribution of astrocytes to the neuroinflammatory response in multiple sclerosis and experimental autoimmune encephalomyelitis
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Tác giả
Roberta Brambilla.
Nội dung
Pages 757-783
5. Seeding selectivity and ultrasensitive detection of tau aggregate conformers of Alzheimer disease
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Tác giả
Allison Kraus, Eri Saijo, Michael A. Metrick II, Kathy Newell.
Nội dung
Pages 585-598
5. Multiple system atrophy prions retain strain specificity after serial propagation in two different Tg(SNCA*A53T) mouse lines
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Tác giả
Amanda L. Woerman, Abby Oehler, Sabeen A. Kazmi, Jisoo Lee.
Nội dung
Pages 437-454
5. TIA1 regulates the generation and response to toxic tau oligomers
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Tác giả
Lulu Jiang, Peter E. A. Ash, Brandon F. Maziuk.
Nội dung
Pages 259-277
5. The intact postsynaptic protein neurogranin is reduced in brain tissue from patients with familial and sporadic Alzheimer’s disease
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Tác giả
Hlin Kvartsberg, Tammaryn Lashley, Christina E. Murray.
Nội dung
Pages 89-102
6. Detrimental and protective action of microglial extracellular vesicles on myelin lesions: astrocyte involvement in remyelination failure
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Tác giả
Marta Lombardi, Roberta Parolisi, Federica Scaroni.
Nội dung
Pages 987-1012
6. Lewy-related pathology exhibits two anatomically and genetically distinct progression patterns: a population-based study of Finns aged 85+
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Tác giả
Anna Raunio, Karri Kaivola, Jarno Tuimala, Mia Kero, Minna Oinas.
Nội dung
Pages 771-782
6. Depopulation of dense α-synuclein aggregates is associated with rescue of dopamine neuron dysfunction and death in a new Parkinson’s disease model
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Tác giả
Michal Wegrzynowicz, Dana Bar-On, Laura Calo’, Oleg Anichtchik.
Nội dung
Pages 575-595
6. Endogenous oligodendroglial alpha-synuclein and TPPP/p25α orchestrate alpha-synuclein pathology in experimental multiple system atrophy models
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Tác giả
Panagiota Mavroeidi, Fedra Arvanitaki.
Nội dung
Pages 415-441
6. A nonsynonymous mutation in PLCG2 reduces the risk of Alzheimer’s disease, dementia with Lewy bodies and frontotemporal dementia, and increases the likelihood of longevity
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Tác giả
Sven J. van der Lee, Olivia J. Conway, Iris Jansen.
Nội dung
Pages 237-250
6. ALS/FTD mutant CHCHD10 mice reveal a tissue-specific toxic gain-of-function and mitochondrial stress response
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Tác giả
Corey J. Anderson, Kirsten Bredvik, Suzanne R. Burstein.
Nội dung
Pages 103-121
6. LRRK2 modifies α-syn pathology and spread in mouse models and human neurons
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Tác giả
Gregor Bieri, Michel Brahic, Luc Bousset, Julien Couthouis.
Nội dung
Pages 961-980
6. The origin, fate, and contribution of macrophages to spinal cord injury pathology
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Tác giả
Lindsay M. Milich, Christine B. Ryan, Jae K. Lee.
Nội dung
Pages 785-797
6. Aggregated Tau activates NLRP3–ASC inflammasome exacerbating exogenously seeded and non-exogenously seeded Tau pathology in vivo
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Tác giả
Ilie-Cosmin Stancu, Niels Cremers, Hannah Vanrusselt.
Nội dung
Pages 599-617
6. Wide distribution of alpha-synuclein oligomers in multiple system atrophy brain detected by proximity ligation
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Tác giả
Hiroaki Sekiya, Hisatomo Kowa, Hinako Koga, Mariko Takata.
Nội dung
Pages 455-466
6. Novel tau fragments in cerebrospinal fluid: relation to tangle pathology and cognitive decline in Alzheimer’s disease
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Tác giả
Claudia Cicognola, Gunnar Brinkmalm, Jessica Wahlgren.
Nội dung
Pages 279-296
6. CorrePicomolar concentrations of oligomeric alpha-synuclein sensitizes TLR4 to play an initiating role in Parkinson’s disease pathogenesis
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Tác giả
Craig D. Hughes, Minee L. Choi, Mina Ryten, Lee Hopkins.
Nội dung
Pages 103-120
7. MSTO1 mutations cause mtDNA depletion, manifesting as muscular dystrophy with cerebellar involvement
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Tác giả
S. Donkervoort, R. Sabouny, P. Yun, L. Gauquelin, K. R. Chao.
Nội dung
Pages 1013-1031
7. Increased prevalence of granulovacuolar degeneration in C9orf72 mutation
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Tác giả
Yuichi Riku, Charles Duyckaerts, Susana Boluda, Isabelle Plu.
Nội dung
Pages 783-793
7. Alzheimer’s disease clinical variants show distinct regional patterns of neurofibrillary tangle accumulation
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Tác giả
Cathrine Petersen, Amber L. Nolan, Elisa de Paula França Resende.
Nội dung
Pages 597-612
7. Vitamin D increases glucocorticoid efficacy via inhibition of mTORC1 in experimental models of multiple sclerosis
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Tác giả
Robert Hoepner, Maud Bagnoud, Maximilian Pistor, Anke Salmen.
Nội dung
Pages 443-456
7. Galectin-3, a novel endogenous TREM2 ligand, detrimentally regulates inflammatory response in Alzheimer’s disease
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Tác giả
Antonio Boza-Serrano, Rocío Ruiz, Raquel Sanchez-Varo.
Nội dung
Pages 251-273
7. Mitochondrial defect in muscle precedes neuromuscular junction degeneration and motor neuron death in CHCHD10S59L/+ mouse
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Tác giả
Emmanuelle C. Genin, Blandine Madji Hounoum, Sylvie Bannwarth.
Nội dung
Pages 123-145
7. Demonstration of prion-like properties of mutant huntingtin fibrils in both in vitro and in vivo paradigms
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Tác giả
Maria Masnata, Giacomo Sciacca, Alexander Maxan, Luc Bousset.
Nội dung
Pages 981-1001
7. Correction to: The origin, fate, and contribution of macrophages to spinal cord injury pathology
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Tác giả
Lindsay M. Milich, Christine B. Ryan, Jae K. Lee.
Nội dung
Pages 799-800
7. Microglial nodules provide the environment for pathogenic T cells in human encephalitis
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Tác giả
Anna R. Tröscher, Isabella Wimmer, Lucía Quemada-Garrido.
Nội dung
Pages 619-635
7. Circulating AQP4-specific auto-antibodies alone can induce neuromyelitis optica spectrum disorder in the rat
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Tác giả
Sophie Hillebrand, Kathrin Schanda, Magdalini Nigritinou.
Nội dung
Pages 467-485
7. Molecular profiling of tumors of the brainstem by sequencing of CSF-derived circulating tumor DNA
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Tác giả
Changcun Pan, Bill H. Diplas, Xin Chen, Yuliang Wu, Xiong Xiao.
Nội dung
Pages 297-306
7. Correction to: Picomolar concentrations of oligomeric alpha-synuclein sensitizes TLR4 to play an initiating role in Parkinson’s disease pathogenesis
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Tác giả
Craig D. Hughes, Minee L. Choi, Mina Ryten, Lee Hopkins.
Nội dung
Page 121
8. The dystroglycan receptor maintains glioma stem cells in the vascular niche
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Tác giả
Bryan W. Day, Justin D. Lathia, Zara C. Bruce.
Nội dung
Pages 1033-1052
8. C9orf72 intermediate repeats are associated with corticobasal degeneration, increased C9orf72 expression and disruption of autophagy
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Tác giả
Christopher P. Cali, Maribel Patino, Yee Kit Tai, Wan Yun Ho.
Nội dung
Pages 795-811
8. Impact of TREM2 risk variants on brain region-specific immune activation and plaque microenvironment in Alzheimer’s disease patient brain samples
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Tác giả
Stefan Prokop, Kelly R. Miller, Sergio R. Labra, Rose M. Pitkin.
Nội dung
Pages 613-630
8. Blood vessels guide Schwann cell migration in the adult demyelinated CNS through Eph/ephrin signaling
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Tác giả
Beatriz Garcia-Diaz, Corinne Bachelin, Fanny Coulpier.
Nội dung
Pages 457-476
8. RhoA regulates translation of the Nogo-A decoy SPARC in white matter-invading glioblastomas
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Tác giả
Peter Wirthschaft, Julia Bode, Himanshu Soni, Fabio Dietrich.
Nội dung
Pages 275-293
8. Ketogenic diet ameliorates axonal defects and promotes myelination in Pelizaeus–Merzbacher disease
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Tác giả
Sina K. Stumpf, Stefan A. Berghoff, Andrea Trevisiol, Lena Spieth.
Nội dung
Pages 147-161
8. Desmoplastic/nodular medulloblastomas (DNMB) and medulloblastomas with extensive nodularity (MBEN) disclose similar epigenetic signatures but different transcriptional profiles
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Tác giả
Andrey Korshunov, Felix Sahm, Konstantin Okonechnikov.
Nội dung
Pages 1003-1015
8. Beta-amyloid pathology in human brain microvessel extracts from the parietal cortex: relation with cerebral amyloid angiopathy and Alzheimer’s disease
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Tác giả
Philippe Bourassa, Cyntia Tremblay, Julie A. Schneider.
Nội dung
Pages 801-823
8. H3.3 K27M depletion increases differentiation and extends latency of diffuse intrinsic pontine glioma growth in vivo
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Tác giả
André B. Silveira, Lawryn H. Kasper, Yiping Fan, Hongjian Jin.
Nội dung
Pages 637-655
8. C9orf72 arginine-rich dipeptide proteins interact with ribosomal proteins in vivo to induce a toxic translational arrest that is rescued by eIF1A
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Tác giả
Thomas G. Moens, Teresa Niccoli, Katherine M. Wilson.
Nội dung
Pages 487-500
8. The impact of histopathology and NAB2–STAT6 fusion subtype in classification and grading of meningeal solitary fibrous tumor/hemangiopericytoma
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Tác giả
Karen Fritchie, Kassandra Jensch, Evgeny A. Moskalev, Alissa Caron.
Nội dung
Pages 307-319
8. Structure and evolution of double minutes in diagnosis and relapse brain tumors
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Tác giả
Ke Xu, Liang Ding, Ti-Cheng Chang, Ying Shao, Jason Chiang.
Nội dung
Pages 123-137
9. Epigenetic loss of RNA-methyltransferase NSUN5 in glioma targets ribosomes to drive a stress adaptive translational program
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Tác giả
Maxime Janin, Vanessa Ortiz-Barahona, Manuel Castro de Moura.
Nội dung
Pages 1053-1074
9. Splicing repression is a major function of TDP-43 in motor neurons
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Tác giả
Aneesh Donde, Mingkuan Sun, Jonathan P. Ling.
Nội dung
Pages 813-826
9. BIN1 recovers tauopathy-induced long-term memory deficits in mice and interacts with Tau through Thr348 phosphorylation
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Tác giả
Maxime Sartori, Tiago Mendes, Shruti Desai, Alessia Lasorsa.
Nội dung
Pages 631-652
9. Impairments in contractility and cytoskeletal organisation cause nuclear defects in nemaline myopathy
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Tác giả
Jacob A. Ross, Yotam Levy, Michela Ripolone, Justin S. Kolb.
Nội dung
Pages 477-495
9. Mutational patterns and regulatory networks in epigenetic subgroups of meningioma
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Tác giả
Nagarajan Paramasivam, Daniel Hübschmann, Umut H Toprak.
Nội dung
Pages 295-308
9. Detection of AD-specific four repeat tau with deamidated asparagine residue 279-specific fraction purified from 4R tau polyclonal antibody
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Tác giả
Momoko Ebashi, Shuta Toru, Ayako Nakamura, Satoshi Kamei.
Nội dung
Pages 163-166
9. Severe bornavirus-encephalitis presenting as Guillain–Barré-syndrome
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Tác giả
Roland Coras, Klaus Korn, Stefanie Kuerten, Hagen B. Huttner.
Nội dung
Pages 1017-1019
9. Naturally occurring antibodies isolated from PD patients inhibit synuclein seeding in vitro and recognize Lewy pathology
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Tác giả
Xinyi Li, Wouter Koudstaal, Lauren Fletcher, Martha Costa.
Nội dung
Pages 825-836
9. TCF4 (E2-2) harbors tumor suppressive functions in SHH medulloblastoma
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Tác giả
Malte Hellwig, Marlen C. Lauffer, Michael Bockmayr, Michael Spohn.
Nội dung
Pages 657-673
9. ACTN2 mutations cause “Multiple structured Core Disease” (MsCD)
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Tác giả
Xavière Lornage, Norma B. Romero, Claire A. Grosgogeat.
Nội dung
Pages 501-519
9. Microglial cell loss after ischemic stroke favors brain neutrophil accumulation
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Tác giả
Amaia Otxoa-de-Amezaga, Francesc Miró-Mur, Jordi Pedragosa.
Nội dung
Pages 321-341
9. The genetic landscape of gliomas arising after therapeutic radiation
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Tác giả
Giselle Y. López, Jessica Van Ziffle, Courtney Onodera.
Nội dung
Pages 139-150
10. MYCN amplification drives an aggressive form of spinal ependymoma
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Tác giả
David R. Ghasemi, Martin Sill, Konstantin Okonechnikov.
Nội dung
Pages 1075-1089
10. Routine RNA sequencing of formalin-fixed paraffin-embedded specimens in neuropathology diagnostics identifies diagnostically and therapeutically relevant gene fusions
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Tác giả
Damian Stichel, Daniel Schrimpf, Belén Casalini, Jochen Meyer.
Nội dung
Pages 827-835
10. The neuropathology of fatal encephalomyelitis in human Borna virus infection
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Tác giả
Friederike Liesche, Viktoria Ruf, Saida Zoubaa, Gwendolyn Kaletka.
Nội dung
Pages 653-665
10. Rosette-forming glioneuronal tumors share a distinct DNA methylation profile and mutations in FGFR1, with recurrent co-mutation of PIK3CA and NF1
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Tác giả
Philipp Sievers, Romain Appay, Daniel Schrimpf, Damian Stichel.
Nội dung
Pages 497-504
10. Second-generation molecular subgrouping of medulloblastoma: an international meta-analysis of Group 3 and Group 4 subtypes
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Tác giả
Tanvi Sharma, Edward C. Schwalbe, Daniel Williamson, Martin Sill.
Nội dung
Pages 309-326
10. Revisiting the utility of TDP-43 immunoreactive (TDP-43-ir) pathology to classify FTLD-TDP subtypes
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Tác giả
Yasushi Nishihira MD, PhD, Tamar Gefen PhD, Qinwen Mao.
Nội dung
Pages 167-169
10. Correction to: H3.3 K27M depletion increases differentiation and extends latency of diffuse intrinsic pontine glioma growth in vivo
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André B. Silveira, Lawryn H. Kasper, Yiping Fan, Hongjian Jin.
Nội dung
Page 1021
10. Papillary glioneuronal tumor (PGNT) exhibits a characteristic methylation profile and fusions involving PRKCA
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Yanghao Hou, Jorge Pinheiro, Felix Sahm, David E. Reuss.
Nội dung
Pages 837-846
10. SHH medulloblastoma in a young adult with a TCF4 germline pathogenic variation
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Maud Blanluet, Julien Masliah-Planchon, Irina Giurgea.
Nội dung
Pages 675-678
10. Primary intracranial sarcomas with DICER1 mutation often contain prominent eosinophilic cytoplasmic globules and can occur in the setting of neurofibromatosis type 1
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Julieann C. Lee, Javier E. Villanueva-Meyer, Sean P. Ferris.
Nội dung
Pages 521-525
10. Inner ear pathologies impair sodium-regulated ion transport in Meniere’s disease
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Andreas H. Eckhard, MengYu Zhu, Jennifer T. O’Malley.
Nội dung
Pages 343-357
10. Rapid lymphatic efflux limits cerebrospinal fluid flow to the brain
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Qiaoli Ma, Miriam Ries, Yann Decker, Andreas Müller, Chantal Riner…
Nội dung
Pages 151-165
11. A single-center study of the clinicopathologic correlates of gliomas with a MYB or MYBL1 alteration
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Jason Chiang, Julie H. Harreld, Christopher L. Tinkle.
Nội dung
Pages 1091-1092
11. Restoring brain cholesterol turnover improves autophagy and has therapeutic potential in mouse models of spinocerebellar ataxia
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Clévio Nóbrega, Liliana Mendonça, Adriana Marcelo.
Nội dung
Pages 837-858
11. Dipeptide repeat (DPR) pathology in the skeletal muscle of ALS patients with C9ORF72 repeat expansion
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Matthew D. Cykowski, Dennis W. Dickson, Suzanne Z. Powell.
Nội dung
Pages 667-670
11. Molecular progression of SHH-activated medulloblastomas
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Andrey Korshunov, Konstantin Okonechnikov, Felix Sahm.
Nội dung
Pages 327-330
11. Two molecularly distinct atypical teratoid/rhabdoid tumors (or tumor components) occurring in an infant with rhabdoid tumor predisposition syndrome 1
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Christian Thomas, Friederike Knerlich-Lukoschus, Harald Reinhard.
Nội dung
Pages 847-850
11. Prognostic significance of NAB2–STAT6 fusion variants and TERT promotor mutations in solitary fibrous tumors/hemangiopericytomas of the CNS: not (yet) clear
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Rob Vogels, Nicolas Macagno, Klaus Griewank, Patricia Groenen.
Nội dung
Pages 679-682
11. Antisense RNA foci are associated with nucleoli and TDP-43 mislocalization in C9orf72-ALS/FTD: a quantitative study
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Olubankole Aladesuyi Arogundade, Jennifer E. Stauffer.
Nội dung
Pages 527-530
11. Enteric alpha-synuclein expression is increased in Crohn’s disease
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Alice Prigent, Arthur Lionnet, Emilie Durieu, Guillaume Chapelet.
Nội dung
Pages 359-361
11. “Minimal change” multiple system atrophy with limbic-predominant α-synuclein pathology
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Tác giả
Shunsuke Koga, Dennis W. Dickson.
Nội dung
Pages 167-169
12. C9orf72-specific phenomena associated with frontotemporal dementia and gastrointestinal symptoms in the absence of TDP-43 aggregation
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Tác giả
Paul J. Sampognaro, Sarat C. Vatsavayai, Celica G. Cosme.
Nội dung
Pages 1093-1097
12. Contextualizing the pathology in the essential tremor cerebellar cortex: a patholog-omics approach
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Elan D. Louis, Chloë A. Kerridge, Debotri Chatterjee.
Nội dung
Pages 859-876
12. Controversies about the subcellular localization and mechanisms of action of the Alzheimer's disease-protective CD33 splice variant
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Sudeshna Saha, Shoib S. Siddiqui, Naazneen Khan, Andrea Verhagen.
Nội dung
Pages 671-672
12. Lack of H3K27 trimethylation is associated with 1p/19q codeletion in diffuse gliomas
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Tác giả
Katharina Filipski, Yannick Braun, Jenny Zinke, Bastian Roller.
Nội dung
Pages 331-334
12. Recurrent KBTBD4 small in-frame insertions and absence of DROSHA deletion or DICER1 mutation differentiate pineal parenchymal tumor of intermediate differentiation (PPTID) from pineoblastoma
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Tác giả
Julieann C. Lee, Tali Mazor, Richard Lao, Eunice Wan.
Nội dung
Pages 851-854
12. cIMPACT-NOW update 4: diffuse gliomas characterized by MYB, MYBL1, or FGFR1 alterations or BRAFV600E mutation
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Tác giả
David W. Ellison, Cynthia Hawkins, David T. W. Jones.
Nội dung
Pages 683-687
12. Atypical parkinsonism of progressive supranuclear palsy–parkinsonism (PSP-P) phenotype with rare variants in FBXO7 and VPS35 genes associated with Lewy body pathology
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Tác giả
Kateřina Menšíková, Lucie Tučková, Kristýna Kolařiková.
Nội dung
Pages 171-173
13. Production of poly(GA) in C9ORF72 patient motor neurons derived from induced pluripotent stem cells
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Tác giả
Sandra Almeida, Gopinath Krishnan, Mia Rushe, Yuanzheng Gu.
Nội dung
Pages 1099-1101
13. Recurrent non-canonical histone H3 mutations in spinal cord diffuse gliomas
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Tác giả
Emily A. Sloan, Tabitha Cooney, Nancy Ann Oberheim Bush.
Nội dung
Pages 877-881
13. Correction to: Ketogenic diet ameliorates axonal defects and promotes myelination in Pelizaeus–Merzbacher disease
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Tác giả
Sina K. Stumpf, Stefan A. Berghoff, Andrea Trevisiol, Lena Spieth.
Nội dung
Pages 673-674
13. ALK-positive histiocytosis with KIF5B-ALK fusion in the central nervous system
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Tác giả
Calixto-Hope G. Lucas, Ahmed Gilani, David A. Solomon.
Nội dung
Pages 335-337
13. An ALS case with 38 (G4C2)-repeats in the C9orf72 gene shows TDP-43 and sparse dipeptide repeat protein pathology
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Tác giả
Lieselot Dedeene, Evelien Van Schoor, Valérie Race.
Nội dung
Pages 855-858
13. ETMR-like infantile cerebellar embryonal tumors in the extended morphologic spectrum of DICER1-related tumors
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Tác giả
Emmanuelle Uro-Coste, Julien Masliah-Planchon, Aurore Siegfried.
Nội dung
Pages 175-177
14. Neuroglial stem cell-derived inflammatory pseudotumor (n-SCIPT): clinicopathologic characterization of a novel lesion of the lumbosacral spinal cord and nerve roots following intrathecal allogeneic stem cell intervention
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Tác giả
Emily A. Sloan, Paul J. Sampognaro, Jacqueline C. Junn.
Nội dung
Pages 1103-1106
14. Correction to: ADAR2 mislocalization and widespread RNA editing aberrations in C9orf72-mediated ALS/FTD
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Stephen Moore, Eric Alsop, Ileana Lorenzini, Alexander Starr.
Nội dung
Pages 883-884
14. Correction to: Endogenous oligodendroglial alpha-synuclein and TPPP/p25α orchestrate alpha-synuclein pathology in experimental multiple system atrophy models
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Panagiota Mavroeidi, Fedra Arvanitaki.
Nội dung
Page 675
14. Chromosome arm 1q gain is an adverse prognostic factor in localized and diffuse leptomeningeal glioneuronal tumors with BRAF gene fusion and 1p deletion
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Tác giả
Jason Chiang, James Dalton, Santhosh A. Upadhyaya, Zoltán Patay.
Nội dung
Pages 179-181
