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Basic Research in Cardiology
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1. Correction to: Exercise-induced circulating extracellular vesicles protect against cardiac ischemia–reperfusion injury
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Tác giả
Yihua Bei, Tianzhao Xu, Dongchao Lv, Pujiao Yu, Jiahong Xu.
1. Connexin 43 dephosphorylation contributes to arrhythmias and cardiomyocyte apoptosis in ischemia/reperfusion hearts
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Tác giả
Jingyi Xue, Xinxin Yan, Yutong Yang, Min Chen, Lulin Wu.
1. In memoriam: John Ross, Jr
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Tác giả
Gianluigi Condorelli, Gerd Heusch.
1. Reduced ambient temperature exacerbates SIRS-induced cardiac autonomic dysregulation and myocardial dysfunction in mice
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Tác giả
Bernadin Ndongson-Dongmo, Guang-Ping Lang, Odeta Mece.
1. Protein kinase/phosphatase balance mediates the effects of increased late sodium current on ventricular calcium cycling
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Tác giả
Jörg Eiringhaus, Jonas Herting, Felix Schatter.
1. Inflammatory cells and their non-coding RNAs as targets for treating myocardial infarction
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Tác giả
Mira Jung, Michael Dodsworth, Thomas Thum.
2. Enhancement of cardiac lymphangiogenesis by transplantation of CD34+VEGFR-3+ endothelial progenitor cells and sustained release of VEGF-C
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Tác giả
Hai-feng Zhang, Yong-li Wang, Yu-zhen Tan, Hai-jie Wang.
2. Cardioprotection of post-ischemic moderate ROS against ischemia/reperfusion via STAT3-induced the inhibition of MCU opening
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Tác giả
Lan Wu, Ji-Liang Tan, Zhong-Yan Chen, Gang Huang.
2. Relaxin mitigates microvascular damage and inflammation following cardiac ischemia–reperfusion
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Tác giả
Xiao-Ming Gao, Yidan Su, Shirley Moore, Li-Ping Han.
2. Inhibition of sodium–glucose cotransporter-2 preserves cardiac function during regional myocardial ischemia independent of alterations in myocardial substrate utilization
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Tác giả
Hana E. Baker, Alexander M. Kiel, Samuel T. Luebbe.
2. The different response of cardiomyocytes and cardiac fibroblasts to mitochondria inhibition and the underlying role of STAT3
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Tác giả
Jing Zhao, Jin-Lai Gao, Jun-Xue Zhu, Hai-Bin Zhu, Xuan Peng.
2. Epigenetically modified cardiac mesenchymal stromal cells limit myocardial fibrosis and promote functional recovery in a model of chronic ischemic cardiomyopathy
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Tác giả
Joseph B. Moore IV, Xian-Liang Tang, John Zhao.
3. Junctophilin-2 is a target of matrix metalloproteinase-2 in myocardial ischemia–reperfusion injury
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Tác giả
Brandon Y. H. Chan, Andrej Roczkowsky, Woo Jung Cho.
3. Mitochondrial bioenergetics links inflammation and cardiac contractility in endotoxemia
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Tác giả
Tamara Antonela Vico, Timoteo Marchini, Santiago Ginart.
3. P66shc and its role in ischemic cardiovascular diseases
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Tác giả
Kerstin Boengler, Julia Bornbaum, Klaus-Dieter Schlüter.
3. Correction to: Protection against cardiac ischemia-reperfusion injury by hypothermia and by inhibition of succinate accumulation and oxidation is additive
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Tác giả
M. Kohlhauer, V. R. Pell, N. Burger, A. M. Spiroski.
3. Blocking the IL-1β signalling pathway prevents chronic viral myocarditis and cardiac remodeling
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Tác giả
Lisa Kraft, Tugs Erdenesukh, Martina Sauter.
3. Of mice and men: models and mechanisms of diabetic cardiomyopathy
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Tác giả
Christian Riehle, Johann Bauersachs.
4. Carotid baroreceptor stimulation suppresses ventricular fibrillation in canines with chronic heart failure
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Tác giả
Jing Wang, Mingyan Dai, Quan Cao, Qiao Yu, Qiang Luo.
4. Neutrophil proteome shifts over the myocardial infarction time continuum
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Tác giả
Michael J. Daseke II, Fritz M. Valerio, William J. Kalusche.
4. E2f1 deletion attenuates infarct-induced ventricular remodeling without affecting O-GlcNAcylation
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Tác giả
Sujith Dassanayaka, Kenneth R. Brittian, Andrea Jurkovic.
4. Authorship: from credit to accountability. Reflections from the Editors’ Network
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Tác giả
Fernando Alfonso, Parounak Zelveian, Jean-Jacques Monsuez.
4. Pericardial fluid: an underrated molecular library of heart conditions and a potential vehicle for cardiac therapy
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Tác giả
Fábio Trindade, Rui Vitorino, Adelino Leite-Moreira.
4. Elevated monocyte-specific type I interferon signalling correlates positively with cardiac healing in myocardial infarct patients but interferon alpha application deteriorates myocardial healing in rats
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Tác giả
Ellis N. ter Horst, Paul A. J. Krijnen, Nazanin Hakimzadeh.
5. Therapies to limit myocardial injury in animal models of myocarditis: a systematic review and meta-analysis
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Tác giả
Joshua A. Silverblatt, Oliver J. Ziff, Luke Dancy.
5. Mono- and multi-nucleated ventricular cardiomyocytes constitute a transcriptionally homogenous cell population
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Tác giả
Michail Yekelchyk, Stefan Guenther, Jens Preussner.
5. Atrial fibrillation is associated with alterations in HDL function, metabolism, and particle number
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Tác giả
Markus Trieb, Jelena Kornej, Eva Knuplez, Gerhard Hindricks.
5. Attenuation of ST-segment elevation after ischemic conditioning maneuvers reflects cardioprotection online
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Tác giả
Georgios Amanakis, Petra Kleinbongard, Gerd Heusch.
5. Repeated cell transplantation and adjunct renal denervation in ischemic heart failure: exploring modalities for improving cell therapy efficacy
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Tác giả
David J. Polhemus, Rishi K. Trivedi, Thomas E. Sharp.
6. Glucocorticoids preserve the t-tubular system in ventricular cardiomyocytes by upregulation of autophagic flux
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Tác giả
Thomas Seidel, Dominik J. Fiegle, Tim J. Baur, Anne Ritzer.
6. DPP-4 inhibition by linagliptin prevents cardiac dysfunction and inflammation by targeting the Nlrp3/ASC inflammasome
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Tác giả
Yochai Birnbaum, Dat Tran, Mandeep Bajaj, Yumei Ye.
6. Calpains mediate isoproterenol-induced hypertrophy through modulation of GRK2
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Tác giả
David Aluja, Javier Inserte, Petronila Penela, Paula Ramos.
6. Endothelial α1AMPK modulates angiotensin II-mediated vascular inflammation and dysfunction
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Tác giả
Swenja Kröller-Schön, Thomas Jansen, Thi Lan P. Tran.
7. Acute exposure to nocturnal train noise induces endothelial dysfunction and pro-thromboinflammatory changes of the plasma proteome in healthy subjects
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Tác giả
Johannes Herzog, Frank P. Schmidt, Omar Hahad.
7. Reparative macrophage transplantation for myocardial repair: a refinement of bone marrow mononuclear cell-based therapy
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Tác giả
Mihai-Nicolae Podaru, Laura Fields, Satoshi Kainuma.
7. Exogenous GDF11 attenuates non-canonical TGF-β signaling to protect the heart from acute myocardial ischemia–reperfusion injury
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Tác giả
Hsing-Hui Su, Jiuan-Miaw Liao, Yi-Hsin Wang, Ke-Min Chen.
7. Inhibition of cardiomyocyte Sprouty1 protects from cardiac ischemia–reperfusion injury
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Tác giả
Tarja Alakoski, Johanna Ulvila, Raisa Yrjölä, Laura Vainio.
8. Coronary microvascular obstruction: the new frontier in cardioprotection
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Tác giả
Gerd Heusch.
8. Neutrophil extracellular traps and fibrocytes in ST-segment elevation myocardial infarction
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Tác giả
Thomas M. Hofbauer, Andreas Mangold, Thomas Scherz.
8. Inflammation and fibrosis in murine models of heart failure
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Tác giả
Lucas Bacmeister, Michael Schwarzl, Svenja Warnke.
8. Fibroblast polarization over the myocardial infarction time continuum shifts roles from inflammation to angiogenesis
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Tác giả
Alan J. Mouton, Yonggang Ma, Osvaldo J. Rivera Gonzalez.
9. Inducible cardiac-specific overexpression of cyclooxygenase-2 (COX-2) confers resistance to ischemia/reperfusion injury
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Tác giả
Yiru Guo, Yibing Nong, Deepali Nivas Tukaye, Gregg Rokosh.
9. Protection against cardiac ischemia-reperfusion injury by hypothermia and by inhibition of succinate accumulation and oxidation is additive
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Tác giả
M. Kohlhauer, V. R. Pell, N. Burger, A. M. Spiroski.
9. Effect of pulmonary artery denervation in postcapillary pulmonary hypertension: results of a randomized controlled translational study Ines
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Tác giả
Garcia-Lunar, Daniel Pereda, Evelyn Santiago.
10. Increased calpain-1 in mitochondria induces dilated heart failure in mice: role of mitochondrial superoxide anion
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Tác giả
Ting Cao, Shuai Fan, Dong Zheng, Grace Wang, Yong Yu.
11. Things get broken: the hypoxia-inducible factor prolyl hydroxylases in ischemic heart disease
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Tác giả
Timm Schreiber, Luca Salhöfer, Theresa Quinting.
12. Acute and chronic remote ischemic conditioning attenuate septic cardiomyopathy, improve cardiac output, protect systemic organs, and improve mortality in a lipopolysaccharide-induced sepsis model
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Tác giả
Takashi Honda, Quan He, Fangfei Wang, Andrew N. Redington.
13. Remote ischaemic preconditioning ameliorates sinus rhythm restoration rate through Cox maze radiofrequency procedure associated with inflammation reaction reduction
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Tác giả
Qin Jiang, Bo Xiang, Haitao Wang, Keli Huang, Hong Kong.